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Activation of the GLP-1 receptor (Figure 4) may directly or indirectly inhibit NKκB transcription through the attenuation of NF-κB p65 expression and the blocking of IκBα degradation [123,187]
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Gout occurred in 7.4 percent of GLP-1 users, compared with 6.6 percent of those not taking the drugs — about a 12 percent increase
